The scission products further decomposed into lower molecular weight products with drip, which caused the combustion under specimens. There were no big differences in a weight loss temperature and scission products at high temperature between a low

molecular weight of polyethylene and a high molecular weight of polyethylene. selleckchem Therefore, the degradation and combustion behavior of scission products of polyethylene in primary degradation warrant further research on flame retardant of polyethylene. (c) 2011 Wiley Periodicals, Inc. J Appl Polym Sci 122: 436-443, 2011″
“Structural, transport, and magnetoelectric (ME) properties of delafossite oxides CuCr1-xO2 with 0 <= x <= 0.10 were extensively investigated. The Rietveld refinement shows that the Cu-O bond length decreases with increasing Cr deficiency, indicative of the presence of a mixed valence state of Cu+/Cu2+ and an enhancement of the hybridization between Cu 3d and O 2p orbitals. As a result, it leads to a decrease of room-temperature https://www.selleckchem.com/products/cbl0137-cbl-0137.html resistivity by two orders of magnitude. The deduced effective moment for the Cr-deficient samples is larger than the one only taking into account the contribution from Cr3+ with S = 3/2. This demonstrates that Cu2+ is present in the

Cr-deficient samples, giving rise to excess holes at the Cu site. Below T-N(Cr) similar to 24 K, the magnetocapacitance [epsilon(H)-epsilon(0)]/epsilon(0) exhibits a distinct field dependence and deviates from the square of magnetization M-2. These findings suggest that the ME coupling in CuCr1-xO2 with higher x is modulated by an increase of the spin fluctuations in the Stem Cells & Wnt inhibitor CrO2 triangular lattice through the interplay

between charge and spin degrees of freedom. (C) 2011 American Institute of Physics. [doi:10.1063/1.3544498]“
“Medullary thyroid carcinoma (MTC) accounts for 5-8% of all thyroid cancers. MTC is mainly sporadic in nature, but an hereditary pattern [multiple endocrine neoplasia type 2 (MEN 2)] is present in 20-30% of cases, transmitted as an autosomal-dominant trait due to germline mutations of the RET proto-oncogene. About 98% of patients with MEN 2 have germline mutations in exons 5, 8, 10, 11, 13, 14, 15 or 16 of the RET gene. The primary treatment of both hereditary and sporadic forms of MTC is total thyroidectomy and removal of all neoplastic tissue present in the neck. The therapeutic option for lymph node surgery should be dictated by the results of presurgical evaluation. After total thyroidectomy, measurements of serum calcitonin (CT) and carcinoembryonic antigen are of paramount importance in the postsurgical follow-up of patients with MTC as they reflect the presence of persistent or recurrent disease. Complete remission is demonstrated by undetectable and stimulated serum CT measurement.